Hyperbaric Oxygen Treatment Brain Injurychronic Stroke Rehabilitation Part 2Written by admin on April 7th, 2010
In another PET scan study of 31 patients with infarcts involving the frontal sensorimotor cortex, 23 had persistent diaschisis up to 5 years after onset while the remaining 8 had the diaschisis recover without recovery of oxygen metabolism in the infarcted area (implying that tissue in the ischemic penumbra did recover and this is what allowed for recovery of the diaschisis). (Miuura H; et al.1994.)
Thus if functionless ischemic penumbral tissue can be “”re-activated”" and be made to function again, a corresponding amount of the areas of diaschisis will be returned to normal with normal blood flow and function returning.
In a number of studies in normal dogs, monkeys and Man, hyperbaric oxygen has been shown to diminish cerebral blood flow from 1 to 29% (average 14.7%) which some people have claimed to be detrimental to a stroke or brain injured patient. All of these studies were done in normal non-brain injured subjects while the studies that were done in brain injured patients all showed an increase in cerebral blood flow (Jain, 1996 page 239).
Dr. K.K. Jain states, “”Vasoconstriction and reduced cerebral blood flow do not produce any clinically observable effects in a healthy adult when pressures of 1.5 to 2 ATA are used…The effects of HBO are more pronounced in hypoxic/ischemic states of the brain. HBO reduces cerebral edema and improves the function of neurons rendered inactive by ischemia/hypoxia. The improvement of brain function is reflected by the improved electrical activity of the brain.”"
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